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The increase in IGFBP-1 and -2 leads to secondary increases in circulating IGF-I binding capacity that results in high total IGF-I, but decreased IGF-I bioactivity (77, 78) ( Figure 4 )

Supporting this hypothesis, murine studies using an IMQ-triggered psoriasis model revealed an inverse relationship between the serum concentrations of acetate and propionate, and cytokines IL-23/IL-27

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Additionally, serine-derived metabolites such as glutathione, glycine, cysteine, and SAM support phase II metabolism because they are utilized for conjugation with toxins for toxin metabolism and excretion