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Our results indicate increased DNA damage and reduced DNA repair are key targets of the dual agent treatment, and together these suggest the cancer cells are unable to repair increased DNA damage leading to higher cell death, further supporting the potential use of this dual agent as TNBC therapeutic
The occurrence of oxidative stress during primary AHR is interesting, since inflammatory cells have not yet been recruited to the lungs as a result of prior respiratory allergen challenge (27,28)
Clin Cosmet Invest Dermatol
In addition, epithalon delivered by injection may occasionally cause inflammatory reactions surrounding the injection site
