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On the other hand, when A and C fibers form synaptic connections within the dorsal horn, they release a triad of neurotransmitters (glutamate, calcitonin gene-related peptide CGRP, and substance P), thereby initiating the cascade of pain signal transmission [138]
Large animal models of traumatic brain injury
Pain behaviors in these rats and the time course of mechanical hyperalgesia are similar to patients pain reports in the postoperative period [12]
