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Neuroprotection by deprenyl and other propargylamines: glyceraldehyde-3-phosphate dehydrogenase rather than monoamine oxidase B
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Recolonization with a microbiota comparable to before treatment occurs in treated areas
Neurotoxic Triad Excitotoxicity, Oxidative Stress and Neuroinflammation Excitotoxicity occurs when abnormally high levels of glutamate are in the synaptic cleft between neurons, leading to prolonged activation of glutamate receptors, such as the NMDA receptor, resulting in increased calcium influx into the postsynaptic neuron and resultant cell death (Olney, 1990)
