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Genet 19 , 5162 (2018)
Research models show it drives angiogenesis via VEGFR2 signaling and modulates nitric oxide synthesis, accelerating soft-tissue repair at the injury site
Additionally, metabolic profiling has revealed that suppression of GLS1 induces a compensatory anaplerotic mechanism via pyruvate carboxylase (PC), which allows the release of a glutamine-independent supply of TCA intermediates by catalyzing the transformation of pyruvate to oxaloacetate 153
Recent preclinical and clinical data support the promise of several agents
