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This highlights the intricate relationship between epilepsy and mitochondrial dysfunction, emphasizing the need to unravel the multifaceted impact of mitochondrial function on brain activity as a potential avenue for epilepsy treatment
10.1002/jimd.12070
Future studies should investigate whether mitochondria-targeted interventions (e.g., exercise, NAD + precursors, mitophagy inducers) can simultaneously reduce frailty and prevent cardiometabolic-cognitive decline [40]
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