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APS attenuated the increase in cardiomyocyte surface area, improved cardiomyocyte viability, enhanced cardiac function, and reduced the heart weight-to-body weight (HW/BW) ratio in DCM rats

However, acetaminophen toxicity leads to the formation and accumulation of excessive amounts of NAPQI, the depletion of GSH stores, oxidative stress, and mitochondrial dysfunction resulting in the depletion of adenosine triphosphate (ATP) stores [44]

High IGF-1 levels could compete for binding to the IGF-1R thus reversing the inhibitory effect of IGF-1R-targeted antibodies [84]

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