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Petrillo et al., 2019)
10.1006/abio.1999.4047
This effect can be completely reversed by SOD2 expression, indicating that hyperglycemia-induced OXTR suppression is due to hyperglycemia-induced consistent oxidative stress, which has been termed hyperglycemia memory ( in vivo study, which showed that OXTR expression was suppressed in many brain tissues, including the amygdala, hypothalamus and hippocampus, in prenatal diabetes exposure-induced offspring
Abdel Shaheed C, Ferreira GE, Dmitritchenko A, McLachlan AJ, Day RO, Saragiotto B, et al
