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Although there is an emerging role of central glutamine metabolism in exercise, the relative contributions of the GGC and its molecular machinery (GS, system N 1, SAT1, GLS and glutamic acid decarboxylase) to enhance exercise performance, including exercise-induced regulation of neuronal function and/or central fatigue remains unclear

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In terms of IL-1 production, LPS-stimulated BV-2 cells pretreated with 100 and 1000 M of -EC or GSH showed a significant reduction compared to the control (Fig