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However, MenSCs display a significantly faster proliferation rate, with a population doubling time of approximately 19.4 hoursroughly twice as fast as that of bone marrow-derived MSCs (BM-MSCs) (Skliut et al., 2021)

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Lastly, oxidative stress and mitochondrial dysfunction, prominently arising from dopamine autooxidation and impaired mitophagy pathways (e.g., mutations in PINK1, PRKN), are central to neuronal degeneration in PD, emphasizing their importance as therapeutic targets [22, 23]