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In conclusion, various endogenous and exogenous pathophysiological mechanisms in patients with chronic kidney diseases lead to enhanced activity of oxidative enzymes such as xanthine oxidase (XO), myeloperoxidase (MPO), and the NADPH oxidase enzymes (NOX)

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Flavonoids also modulate gene expression by targeting transcription factors like NFB, GATA3, and STAT6, resulting in decreased transcription of proinflammatory genes