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Abnormal forms of EGLN1 interfere with normal HIF degradation, and thus produce inappropriately elevated EPO levels and erythrocytosis

1 Introduction The tumor suppressor protein p53 is often regarded as the gatekeeper of the cell because it is essential for stabilizing the cellular genome ( TP53 , leading to inappropriate activity of wild-type p53 and unrestrained tumor progression ( In the past, apoptosis was regarded as the primary cell death mechanism in conventional cancer treatments

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