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In general, viral infections may contribute to the development of fibrosis in SG through various mechanisms, including the induction of cytokines, chemokines, and interaction with immune cells
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However, in this study, the abundance of immune cells was negatively correlated with the expression of the PPARGC1A gene
The selective self-assembly and uptake of Psa-AR and P-AR were further confirmed by IF, which revealed low cellular uptake of both Psa-AR and P-AR in RWPE-1 cells (Supplementary Fig
