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The pathological characteristics of ALI involve extensive inflammation resulting in harm to alveolar epithelial cells (AECs), breakdown of the pulmonary vascular endothelial barrier and subsequent accumulation of protein-rich inflammatory edema fluid within the alveolar spaces, ultimately causing diffuse alveolar damage (6)

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Coexpressed catalase protects chimeric antigen receptorredirected T cells as well as bystander cells from oxidative stressinduced loss of antitumor activity

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