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This protein kinase is a downstream target of oxidative stress and proinflammatory cytokines and its activation can increase vascular permeability and cell death, the key elements of diabetic retinopathy pathogenesis [142]

We next assessed the functional impact of these interactions on the overall N-myristoylation, focusing on the contribution of the NACB C-terminal domain (Fig

Additionally, BHB catabolism increases the coenzyme Q/QH 2 ratio, reducing mitochondrial ROS generation [93,97]

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