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To address this gap, we disrupted lipid catabolism during fasting by impairing key factors involved in triglyceride hydrolysis, coenzyme A conjugation to mitochondrial acyl chains, and the transcriptional activation of -oxidation
Dual roles of apolipoprotein CI in the formation of atherogenic remnants
10.1038/nm.2581 152 SakamotoH.OkamotoK.AokiM.KatoH.KatsumeA.OhtaA.TsukudaT.ShimmaN.AokiY.ArisawaM.KoharaM.SudohM
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