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Description
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Future research could focus on several areas to enhance our knowledge and improve therapeutic strategies for TAO: Exploring the interactions between oxidative stress and other key pathways: Investigating the interactions between oxidative stress and the inflammation, fibrosis, and immune dysregulation that characterize TAO could lead to a more comprehensive understanding of the disease
At the late stage, increased lipophagy releases PUFAs that serve as substrates for lipid peroxidation, whereas the knockdown of RAB7A or PGRMC1 inhibits lipophagy-induced ferroptosis [210, 211]
