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These cellular stress pathways collectively lead to immune cell infiltration, alveolar epithelial damage, and fibrotic remodeling, ultimately impairing gas exchange efficiency
Mitochondria-localized cGAS suppresses ferroptosis to promote cancer progression
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It is known that IGF1R can activate two canonical pathwaysPI3K/AKT pathway and the extracellular signal-regulated kinase (ERK) pathway 22
