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It is also possible that plasma factors in G6PD-normal recipients could improve the survival of G6PD-deficient RBCs (55)
One report indicated that CAV-1 is homologous to the SOCS pseudosubstrate and functions as a negative regulator of STAT signaling, and that CAV-1 prevents the binding of JAKs and STAT proteins to their receptors by binding phosphotyrosine residues of cytokine receptors 22
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