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Furthermore, an absence of acetyl-CoA generated via pyruvate oxidation remodeled the tricarboxylic acid (TCA) cycle and lipid metabolism, with accumulation of two intermediates which could be produced through other pathways other than the physiological TCA cycle upon sepsis, namely succinate and citrate, both of which are pro-inflammatory mediators

doi: 10.1186/s12931-021-01771-3 9 GuoLWuXWuX

doi: 10.1038/s41420-023-01397-y 161 KoliarakisIPsaroulakiANikolouzakisTKKokkinakisMSgantzosMNGoulielmosGet al

Cell autonomous include mutations and damage in ETC complexes (Complex I and III) and in mtDNA, leading to a dysfunctional mitochondria highly-producing ROS