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Redox sensing and signaling associated with reactive oxygen in chloroplasts, peroxisomes and mitochondria

This review interprets HTY-related mechanisms through direct chemical actions such as radical scavenging and metal chelation, indirect cellular associations indicated by alterations in downstream markers (e.g., Nrf2, NF-B, AMPK-related changes), and translational relevance, evidenced when effects become evident at physiologically attainable exposures and/or in human studies