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10.1097/ijg.0000000000002613 13 GalloA
ParunyakulKChuchoiyAKooltueonSPuttagamnerdPSrisuksaiKSantativongchaiPet al
If these components are compromised, dendritic and axonal defects may fail to recover
The idea is supported by maintenance of functional GSH/GSSG levels by Sp in BSO and neurotoxin treated cells as compared to untreated ones which showed a significant decline in the antioxidant ratio probably, due to enhanced formation of glutathionequinones 43
