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Reference Kurata, Suzuki and Takeda1992)

Therefore, therapeutic strategies that target the distinct mitochondrial metabolic features of CSCs and DTPs, such as disrupting the AMPKOXPHOS axis, inhibiting mitophagy, or decoupling metabolic signaling from DNA repair, represent a promising approach to overcoming chemoresistance in CRC

doi: 10.1289/ehp.10423

Based on this information, it is important to consider p K a in association with nucleophilicity of the thiol/thiolate in order to evaluate the tendency toward oxidation