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glutathione colon cancer DLAT sustains redox homeostasis and prevent colorectal from ferroptosis by regulating SLC25A39-mediated mitochondrial transport Glutathione Modulation Reverses the Growth-promoting

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Antibiotic stewardship in the intensive care unit

glutathione colon cancer DLAT sustains redox homeostasis and prevent colorectal from ferroptosis by regulating SLC25A39-mediated mitochondrial transport Glutathione Modulation Reverses the Growth-promoting

10.2337/dc22-2494 163 WuH.EsteveE.TremaroliV.KhanM

glutathione colon cancer DLAT sustains redox homeostasis and prevent colorectal from ferroptosis by regulating SLC25A39-mediated mitochondrial transport Glutathione Modulation Reverses the Growth-promoting

Rocklin, Calif, USA: Prima Publishing

glutathione colon cancer DLAT sustains redox homeostasis and prevent colorectal from ferroptosis by regulating SLC25A39-mediated mitochondrial transport Glutathione Modulation Reverses the Growth-promoting

10.1016/S0002-8703(99)70250-4 32

glutathione colon cancer DLAT sustains redox homeostasis and prevent colorectal from ferroptosis by regulating SLC25A39-mediated mitochondrial transport Glutathione Modulation Reverses the Growth-promoting

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