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Traumatic brain injury causes chronic cortical inflammation and neuronal dysfunction mediated by microglia

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Thus, Trx1 may also serve as a potential diagnostic marker and be useful in prognostic assessments

In conclusion, the in vivo work on Gstp / mice fully demonstrates that GSTP acts as a negative regulator in sepsis pathophysiology, and recombinant GSTP protein treatment to mice may significantly improve survival in mouse CLP sepsis model