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Another study shows that in the hearts of cold-blooded vertebrates, CK bound to myofibrils is necessary for fully activating myosin-ATPase activity, although their energy conversion rate is lower compared to homeothermic animals [103]
Bravo JA, Forsythe P, Chew MV, et al

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In murine models of Ang II-induced AD, Ang II stimulates neutrophils via the angiotensin receptor AT1, leading to the induction of NOX, ROS production, and adhesion of mononuclear cells to endothelial cells (149)
