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Description
NTP 1993)

Its anti-inflammatory effects are mediated by its ability to suppress the expression and release of pro-inflammatory cytokines, such as Interleukin-6 (IL-6) and Tumor Necrosis Factor-alpha (TNF-), in various cell culture models

[DOI] [PMC free article] [PubMed] [Google Scholar] 22.Park JR, Lee H, Kim SI, et al

This suppression of CYP1A2 is relevant, as it metabolizes asthmatic and psychotic drugs, potentially increasing the risk of drug toxicity ( For instance, Mongolian gerbil studies have reported that quinestrol, an exogenous estrogen, modifies metabolizing enzymes
