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Chen et al., 2015)
PrP suppression ameliorates the phenotype of Atp7b knockout mice To test the pathological relevance of the PrP-mediated mechanism in vivo, we used Atp7b / mice, a well-characterized WD mouse model
doi: 10.1002/bies.201700012 74
In the liver, a major site of GSH metabolism, this consists of: a) extensive catabolism of GSH within biliary spaces (Ballatori et al., 1986b, 1988, 1989), as well as within sinusoidal compartments of some species (Hinchman and Ballatori, 1990, 1994), b) direct hepatic reabsorption of some of the breakdown products (Ballatori et al., 1986a, 1988
