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It has also been shown that exposure to CM remnants causes endothelial cell apoptosis, as well as an increase in the production of plasmin activator inhibitor-1 (PAI-1), a key regulator of thrombus formation, in endothelial cells (73, 106, 107)

Moreover, the mini-A peptide chaperone also appears to have antioxidant properties, capable of preventing copper-mediated ascorbic acid oxidation [130]
