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One interpretation is that brain iron elevation in AD could be a reactive, possibly protective sequestration mechanism (for instance, immobilizing iron in plaques or ferritin)
suggested that misfolding of SOD1 and the subsequent humoral immune response are frequent events in the elderly [71]
Lavoie, L
Similarly, SF pretreatment protected neural cells against A-fragment cytotoxicity and apoptosis in part through upregulation of Bcl-2 [169]
