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Although this is a remarkable development, killing CD38-positive tumor cells with cytotoxic anti-CD38 antibodies is only one of the potential pharmacological uses of targeting CD38
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The endogenous-acetylcholine-induced VEGF expression in neurons and astrocytes [302], the VEGF signaling system is in the rescuing effect of acetylcholine on NMDA-induced long-lasting hippocampal cell damage [303]
CD8+ T cells from miR34 / mice express decreased levels of CD69 and CD25 and less proliferation with reduced ERK phosphorylation than wild-type cells
