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Therefore, enhanced glutaminolysis might eventually contribute to the initiation and progression of cancer by stimulating cell growth via the mTORC1 pathway and enhancing aerobic glycolysis while disrupting the proper elimination of misfolded proteins, damaged DNA and organelles through the inhibition of autophagy and the DNA damage response 166

69:23092314 [DOI] [PubMed] [Google Scholar] Foyer CH, Lopez-Delgado H, Dat JF, Scott IM (1997) Hydrogen peroxide- and glutathione- associated mechanisms of acclamatory stress tolerance and signalling

10.1111/nph.18437 183 WangC.YangF.ZengW.ChenX.QiuZ.WangQ.et al (2024a)

Complex interplay between metabolism and Cd4(+) T-cell activation, differentiation, and function: A novel perspective for atherosclerosis immunotherapy