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2 Therefore, there is an urgent need to further clarify the molecular mechanism of CRC tumorigenesis and pathogenesis
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Advances in bronchiectasis: endotyping, genetics, microbiome, and disease heterogeneity
Central to the pathogenesis of MASH, hepatic lipid overload occurs with excessive uptake of fatty acids released from the adipose tissue, enhanced de novo lipogenesis from excessive carbohydrate intake or inhibition of fatty acid -oxidation (FAO) 3,4,5
