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Pinsornsak P, Niempoog S
We reasoned such a brake could potentially be a transcriptional regulator and focused on transcription factors that have previously been linked to apoptosis, including STAT1, 2 and 4, RELB, NFB, TP53 and FOXO4 (Fig
A comparative assessment of lengthening followed by end-to-end repair and isograft repair of chronically injured peripheral nerves
10.1136/gut.2003.024828
