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A great amount of Th17cell phenotype has been linked to ASD pathogenesis (Moaaz et al., 2019)

(Left) In liver cells, GLP-1 can mediate cholesterol efflux by acting directly on LXR, or by inducing an increase in ATP-binding cassette transporter A1 (ABCA1) mRNA through Ca2+/calmodulin (CaM)-dependent protein kinase kinase/CAM-dependent protein kinase IV/Prolactin regulatory element binding (CaMKK/CaMKIV/PREB), increasing apolipoprotein AI (apo AI) mRNA and promoter expression, and increasing apo AI secretion, thereby mediating intracellular cholesterol efflux

These results show that BSO reduces viability of BL cell lines, while having no effect on control B cells (Fig

Wrote the paper: LL, YZ, and ZY