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In addition, A/R-induced increases in pMLC were attenuated by pretreatment with LA in these cells, demonstrating the role of LA in the MLC pathway of tight junction regulation
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Overexpressed IGFBP-1 in transgenic mice shows that its increase produces hyperinsulinaemia accompanied by glucose intolerance, and that this process is dependent upon IGFBP-1 phosphorylation statewhich is increased in diabetes [259]
