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This is proceeded by peptide bond formation between -Glu-Cys and the amino acid glycine which results in a reduced form of glutathione, known as GSH (Pophaly et al
hTFR1, absent in WT (left), was observed in B-hTFR1 mice (right) and expressed in brain endothelial cells, suggesting a method for drugs to cross the BBB via hTFR1-mediated transendocytosis
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doi: 10.1158/1055-9965.EPI-21-1440
