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In diffuse midline gliomas, including diffuse intrinsic pontine glioma (DIPG), histone H3.3K27M-related mutations are highly prevalent and are associated with aberrant chromatin accessibility and transcriptional profiles [128]

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Research focused on understanding the molecular pathways of cancer progression and the impact of novel therapeutic interventions is still a key area of focus [283,284,285,286]

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Briefly (Fig

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GHK-Cu is commonly studied in laboratory environments involving extracellular matrix regulation, tissue remodeling pathways, peptide signaling analysis, and cellular repair research

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