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At the biochemical level these events form a causal chain: hydrophobic eugenol first partitions into lipid bilayers, increasing membrane fluidity and permeability and producing rapid K + and ATP leakage with PMF collapse

Whereas expression of the enzymatically inactive mutants did not significantly affect the abundancy of cellular ATP comparing to cells transfected with the empty vector, expression of wild-type Ceg14 led to ~90% reduction

B-GOS did not, however, compromise the main olanzapine antipsychotic mechanism, which is mediated through blockage of serotonin 5-HT2A receptors

CRediT authorship contribution statement Anna Rovira: Writing review & editing, Writing original draft, Validation, Methodology, Investigation, Formal analysis, Conceptualization