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The lameness period (an indicator of joint pain and dysfunction) was significantly shorter in animals receiving AOD-9604 plus hyaluronic acid compared to other treatment groups

Animal toxicology studies lasting up to 9 months (in non-human primates) found no significant organ toxicity, genotoxicity, or reproductive toxicity

Activation of polyamine flux causes an increase in energy expenditure by catalyzing polyamine acetylation to produce acetylpolyamines using acetyl-coA as a substrate [48]

Mechanistic investigations have shown that AOD 9604 increases 3-adrenergic receptor expression at the mRNA level, restoring receptor density in systems where lipolytic responsiveness is impaired