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10.1016/j.cell.2013.09.056 Cell 70 MunroS
2011), lifespan ( n = 100) (Svensson et al
Although it could be theoretically possible to enhance the reverse cholesterol effects of LXRs without targeting hepatic lipogenesis with the use of LXR-specific ligands since most of the hepatic responses are due to activation of LXR, this will be a difficult challenge as the ligand binding pocket in both isoforms has been shown to be nearly identical
