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Description
A 2012 publication by Miller showed that 20% of those tested had a genetic defect in the protein that transports B12 to cells
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mAbs, monoclonal antibodies
Treatment sequencing is critical Improvement followed staged progression: Binding + membrane support Immune modulation + neuroregulation Enhanced binding Antioxidant support Glutathione as a late-stage intervention Effective for residual symptoms once upstream processes were addressed
