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Antioxidant therapeutics in parkinsons disease: current challenges and opportunities
However, formation of the phosphodegron in Nrf2 by GSK-3 is inhibited by stimuli that activate protein kinase B (PKB)/Akt
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The binding of antigen/MHC to the TCR complex results in the phosphorylation and activation of PLC1, which then hydrolyzes phospholipids at the plasma membrane to produce DAG and IP 3
