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Description
A0001 is therefore being developed as a treatment modality for Friedreichs ataxia patients and mitochondrial encephalomyopathy, lactic acidosis, and stroke-like episodes (MELAS) where mitochondrial redox pathologies have been identified (Hawi et al., 2012)
Rahlwes KC
Pharmacological strategies that block PERK, NF-B, or NLRP3 have reduced TMAO induced inflammation in preclinical models, indicating their translatable potential
After reading the title and abstract, these four (04) articles were selected for full reading
