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Metabolism abnormalities induced by exposure to excessive UA are important factors mediating the pathological development of HUA-associated multiple organ complications ( via UA excretion agent benzbromarone therapy ( via regulating transforming growth factor 1, nicotinamide adenine dinucleotide phosphate oxidase and AMP-activated protein kinase mediated reactive oxygen species and inflammation (Madlala et al., 2016

PMID: 39036584 14 Clinical, biochemical, and molecular genetic characteristics of patients with primary carnitine deficiency identified by newborn screening in Shanghai, China

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Carnitines increase plasma levels of adenosine and ATP in humans Vasc Med 1997