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Overexpression of Nrf2 and the vitagene pathways play intrinsic roles in boosting the tumorigenic cascade, as well as prospective therapeutic interventions via inhibitors that stop the proliferation and chemoresistance of brain cancer cells by enhancing apoptosis [107]

synthesized generation 3 dendrimers and evaluated their effects on amyloid beta (A1-42) fibrillation, a key factor in Alzheimers disease

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