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Other likely mechanisms such as chronic immune response in the brain, oxidative and inflammatory damage in the brain, neurotransmitter imbalance (e.g., glutamate and GABA), dysregulation of ApoE (the main cholesterol carrier involved in axonal development), and modification of amyloid processing, leading to A immunoreactivity, hyperphosphorylated tau protein, and loss of NMDA receptors have also been suggested to be involved [108]

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In contrast, 55% of PD patients had Lewy microsomes in the mucosa and submucosa, with the detection of Lewy fibers in submucosal biopsies of the rectum as well as the ascending and descending colon in 2365% of PD patients

Biochem (Moscow) 81:700708 Bulcha JT et al (2021) Viral vector platforms within the gene therapy landscape