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Long-acting IGF-1 analogue - bypasses IGF-binding proteins for direct cell-level signaling
Considering the position of the Gly 1228, we could argue that the mutation G1228D will trigger a steric hindrance with T998 that is tightly constrained by F583 and cannot move easily in the apo and LTC 4 -bound conformations
2021;106(2):E592-601
( In vivo efficacy is evidenced by WFA-loaded MEVs significantly suppressing lung tumor xenograft growth ( Beyond small-molecule delivery, MEVs effectively transport nucleic acid therapeutics
