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10.3390/ijms25137379 120 DuanY.WuX.LiangS.JinF
The resulting lactate-rich, acidic TME further reinforces tumor progression via histone lactylation, an epigenetic modification linking metabolic byproducts to transcriptional reprogramming
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Decreased Level of Sirt1 in AKI NAD + depletion also caused the inhibition of Sirt1 and its downstream target genes, which mediates mitochondrial quality control and renal tubular ATP production level
doi: 10.1007/s13167-021-00257-y
