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Mechanism of action: These treatment modalities inhibit the COMT enzyme, reducing catecholamine levels, including norepinephrine and dopamine degradation

In contrast, inhibiting ceramide synthesis in fat-fed streptozotocin (STZ)-treated rats has been shown to improve endothelial dysfunction by enhancing phosphorylation and NO release via the PI3K/Akt/eNOS pathway

Jarin and colleagues [271] show that although testosterone levels of affirmed female adolescents were reduced from 391.7 ng/dL at baseline to 199.3 ng/dL beyond 6-months of therapy, these levels were still significantly higher than the 29.5 ng/dL of total testosterone in biological females at baseline

This is due to its mechanism of action that specifically targets and reduces visceral fat